TY - CHAP M1 - Book, Section TI - Hypertrophic Obstructive Cardiomyopathy and Cardiac Masses A1 - Wasnick, John D. A1 - Nicoara, Alina Y1 - 2019 N1 - T2 - Cardiac Anesthesia and Transesophageal Echocardiography, 2e AB - Previous chapters discussed how fixed obstructions to blood flowing through the heart can lead to significant morbidity and mortality. Aortic and mitral stenosis are two examples of lesions, which prevent the heart from effectively pumping blood. Hypertrophic obstructive cardiomyopathy (HOCM) represents a specific form of hypertrophic cardiomyopathy (HCM), which also manifests, at rest or with provocation (e.g., elevated heart rate, hypovolemia), with dynamic obstruction of left ventricular outflow tract (LVOT) (Figure 10–1). Dynamic outflow obstruction results in syncope, dyspnea, and, at times, sudden death. Although there is an increased incidence of sudden cardiac death (SCD) in the HCM patient with myocardial wall thickness greater than 30 mm or more, the majority of sudden deaths occur in HCM patients with myocardial wall thicknesses less than 30 mm.1 Consequently, all HCM patients should undergo SCD risk stratification at initial evaluation. The American College of Cardiology Foundation and the American Heart Association (ACCF/AHA) developed extensive guidelines for the evaluation and management of the HCM.Although rare, cardiac tumors and other masses at times interfere with valve function, produce emboli, and dynamically obstruct blood flow through the heart. This chapter will examine these different conditions, which are nonetheless linked by their dynamic ability to prevent the heart from properly functioning. SN - PB - McGraw-Hill Education CY - New York, NY Y2 - 2024/03/28 UR - accessanesthesiology.mhmedical.com/content.aspx?aid=1166980987 ER -